OncoBio Consolider Network

Understanding the Cancer Biology

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Ministerio de Economía y Competitividad


It is also important to highlight that several papers are the result of collaborations between OncoBIO groups, including manuscripts in Nat. Med., Cancer Cell, Nat. Struc. Mol. Biol. and EMBO J among others.

The following table shows the number of published collaborations among OncoBIO groups in the last five years. It is important to note that collaborations among the OncoBIO Consortium are still in progress and will lead to new publications in the next few years. These include for instance three publications in Cell, Nature Genetics or Nature Communications (until 2013) as collaboration between OncoBIO groups. 

Collaborations among OncoBIO Groups



A selection from collaborations (2008-2012)

  • The Ink4/Arf locus is a barrier for iPS cell reprogramming. Nature 460:1136-1139, 2009. [OncoBIO 3, 12]
  • A p53-mediated DNA damage response limits reprogramming to ensure iPS cell genomic integrity. Nature 460:1149-1153, 2009. [OncoBIO 3, 5, 12]
  • Isolation and in vitro expansion of human colonic stem cells. Nature Medicine, 2011, Sep 4. doi: 10.1038/nm.2470. [OncoBIO 2,3]
  • Telomerase reverse transcriptase delays aging in cancer-resistant mice. Cell, 135, 609-622, 2008. [OncoBIO 3, 12]
  • Pancreatitis-induced Inflammation Contributes to Pancreatic Cancer by Inhibiting Oncogene-Induced Senescence. Cancer Cell, 19: 728-739. [OncoBIO 1, 12]
  • Telomeres acquire embryonic stem cell characteristics in induced pluripotent stem cells. Cell Stem Cell 4:141-154, 2009. [OncoBIO 3, 12]
  • Increased telomere fragility and fusions resulting from TRF1 deficiency lead to degenerative pathologies and increased cancer in mice. Genes Dev 23:2060-2075, 2009. [OncoBIO 3, 5]
  • A mammalian microRNA cluster controls DNA methylation and telomere recombination via Rbl2-dependent regulation of DNA methyltransferases. Nat Struct Mol Biology. 15, 268-279, 2008. [OncoBIO 3, 12]
  • Exploiting oncogene-induced DNA damage for the selective killing of Myc-driven tumors. Nature Mol. and Struct. Biology [OncoBIO 1, 5]
  • Metastatic Colonization Requires the Repression of the Epithelial-Mesenchymal Transition Inducer Prrx1. Cancer Cell, 22(6):709-24 (2012) [OncoBIO 4, 9]

About us

The OncoBIO Consolider Network was constituted in December 10th 2007 with the main goal of a better understand of the Cancer Biology.

This coordinated project will provide critical information for the development of new strategic opportunities aimed at improving those diagnostic and therapeutic approaches currently used to treat cancer patients.

Upcoming Events

19 October 2015

Send us your CV

If you are interested in working with us, send your CV indicating the position you want to apply to the e-mail oncobio@oncobio.org

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